CCRN : Clinical Competencies

Study concepts, example questions & explanations for CCRN

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Example Questions

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Example Question #11 : Ccrn

All of the following cranial nerves have a either a sensory or motor function involving the eyes EXCEPT:

Possible Answers:

abducens nerve

trochlear nerve

hypoglossal nerve

oculomotor nerve

Correct answer:

hypoglossal nerve

Explanation:

The trochlear nerve (cranial nerve IV) has a motor function involving eye movement. The oculomotor nerve (cranial nerve III) also has a motor function involving eye movement and contraction of the iris. The abducens nerve (cranial nerve VI) has a sensory function involving eye movement. The hypoglossal nerve (cranial nerve XII) has a motor function involving movement of the tongue.

Example Question #12 : Ccrn

A patient in the ICU presents with an acute disorder of attention, perception, and memory impairment. This is most accurately described as:

Possible Answers:

dementia

attention-deficit-hyperactivity-disorder (ADHD)

delirium

dysthymic disorder

Correct answer:

delirium

Explanation:

Delirium is an acute disorder characterized by attention, perception, and memory impairment. It is often the symptom of an underlying condition such as dementia, medication overdose, substance intoxication, and electrolyte disorders.

Example Question #13 : Ccrn

A patient is suffering from dipsogenic diabetes insipidus. Which of the following correctly characterizes this condition?

Possible Answers:

insufficient insulin production by pancreatic beta-cells

the oral ingestion of excessive amount of water results in a suppression of vasopressin (ADH) release and leads to polyuria.

damage to the posterior pituitary results in a deficiency of vasopressin (ADH)

the kidneys are not adequately responding to vasopressin (ADH)

Correct answer:

the oral ingestion of excessive amount of water results in a suppression of vasopressin (ADH) release and leads to polyuria.

Explanation:

Diabetes insipidus is a condition in which an individual has an insensitivity to or a deficiency of vasopressin (ADH). This is a hormone released by the posterior pituitary that is responsible for increasing water reabsorption in the kidney. There are three types of diabetes insipidus: neurogenic DI, nephrogenic DI, and dipsogenic DI. Neurogenic DI is characterized by damage to the posterior pituitary, which results in a deficiency of vasopressin. Nephrogenic DI is characterized by the kidneys inadequately responding to vasopressin. Dipsogenic DI is characterized by the oral ingestion of excessive amount of water, which results in a suppression of vasopressin release and ultimately leads to polyuria. Diabetes mellitus type 1 results from insufficient insulin production by pancreatic beta-cells.

Example Question #13 : Ccrn

The critical care nurse knows that metabolic acidosis is defined by which of the following parameters?

Possible Answers:

pH < 7.35; and a [HCO3-] > 26 mEq/L

pH < 7.45; and a [HCO3-] < 26 mEq/L

pH < 7.35; and a [HCO3-] < 22 mEq/L

pH > 7.35; and a [HCO3-] > 22 mEq/L

Correct answer:

pH < 7.35; and a [HCO3-] < 22 mEq/L

Explanation:

Metabolic acidosis is one of the four main acid-base disorders. The disorder is characterized by a bloodstream pH < 7.35 and a [HCO3-] < 22 mEq/L. The decrease in bicarbonate ion can be due to an endocrine, gastrointestinal, or renal disorder, as well as a nutritional deficiency.

Example Question #14 : Ccrn

Which of the following is Not one of the four primary acid-base disorders?

Possible Answers:

metabolic alkalosis

all of the given answers represent a primary acid-base disorder

renal tubular acidosis

respiratory acidosis

Correct answer:

renal tubular acidosis

Explanation:

The four main acid-base disorders are: respiratory acidosis, respiratory alkalosis, metabolic acidosis, and metabolic alkalosis. Renal tubular acidosis can lead to metabolic acidosis, but it is not one of the primary classifications of acid-base disorders.

Example Question #15 : Ccrn

Which of the following white blood cells release histamine and attract IgE antibodies to their surface?
I. Basophils
II. Neutrophils
III. Mast cells

Possible Answers:

I and III only

III only

II and III only

I, II, and III

Correct answer:

I and III only

Explanation:

Basophils and Mast cells both stimulate an inflammatory response via the release of histamine and attract IgE antibodies to their surface. The major difference between basophils and Mast cells is that basophils survive only a few days circulating in the blood, while Mast cells can survive for weeks while located in bodily tissues. Neutrophils are phagocytic cells that do release cytokines and other granules, but do not release histamine or attract IgE to their surface.

Example Question #17 : Ccrn

A patient is suffering from neurogenic diabetes insipidus. Which of the following correctly characterizes this condition?

Possible Answers:

the oral ingestion of excessive amount of water results in a suppression of vasopressin (ADH) release and leads to polyuria.

insufficient insulin production by pancreatic beta-cells

the kidneys are not adequately responding to vasopressin (ADH)

damage to the posterior pituitary results in a deficiency of vasopressin (ADH)

Correct answer:

damage to the posterior pituitary results in a deficiency of vasopressin (ADH)

Explanation:

Diabetes insipidus is a condition in which an individual has an insensitivity to or a deficiency of vasopressin (ADH). This is a hormone released by the posterior pituitary that is responsible for increasing water reabsorption in the kidney. There are three types of diabetes insipidus: neurogenic DI, nephrogenic DI, and dipsogenic DI. Neurogenic DI is characterized by damage to the posterior pituitary, which results in a deficiency of vasopressin. Nephrogenic DI is characterized by the kidneys inadequately responding to vasopressin. Dipsogenic DI is characterized by the oral ingestion of excessive amount of water, which results in a suppression of vasopressin release and ultimately leads to polyuria. Diabetes mellitus type 1 results from insufficient insulin production by pancreatic beta-cells.

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